A thoughtful concept

The Metabolic Balance programme is based on more than 10,000 individual rules.

Below are three examples to illustrate the different correlations.

Some of these recommendations and correlations are obvious, but others can only be determined by derivations and interactions.

Diabetes & increased triglycerides at normal glucose levels.

Elevated triglycerides but normal glucose levels may indicate underlying factors towards diabetes allowing measures to be taken.

This can also be seen in a study that was carried out with 439 healthy children and adolescents aged between 4 and 20 years who were divided into 4 groups. The average age was between 11.3 and 12.8 years. The BMI fluctuated between 18.4 (normal weight) and 40.6 (severe overweight).

While all 4 groups had an equally high value for fasting blood sugar, the last group needed almost 4 times more insulin. These are fasting values – so we can well imagine that the insulin level after a meal will increase 8 to 10 times, exhausting the pancreas of each of these children over the years.

Triglycerides were shown to be twice as high in the overweight groups as in the normal weight groups – a very good indicator for overweight and the resulting insulin resistance. Good cholesterol (HDL) was clearly higher in the test subjects who had normal weights (58.5) than in the overweight subjects (39.9). The CRP value (c-reactive protein), an important inflammation indicator, is worth a special mention here. The value of the severely overweight test subjects was 33 x higher in this case.

Cholesterol formation and metabolism

The cholesterol circulating in the blood comes from food and is for the most part produced in the body itself, as it is an important component of the cell membranes. Cholesterol also serves the cells as a starting substance to produce oestrogen, testosterone and other hormones as well as bile acids and vitamin D. If the metabolism is intact, the supply and production of cholesterol are balanced.

The self-production route runs via the metabolism intermediate product Acetyl-CoA, which is produced by the metabolism of fats and proteins ... and especially carbohydrates. If too much Acetyl-CoA is produced because the glycogen storage cells are full and no energy is currently required, it is converted into HMG-CoA (3-hydroxy-3-methylglutaryl-coenzyme A). HMG-CoA is converted into cholesterol and coenzyme Q10 with the help of the enzyme reductase via mevalonic acid.  Coenzyme Q10 is needed in the power plants of the cell, the mitochondria for carbohydrates and fat burning.


If a client receives statins because of highly-increased cholesterol values, not only his (or her) own production of cholesterol will be blocked, but also his own production of coenzyme Q10 – and this contributes to a slower metabolism.

Reducing foods containing cholesterol (meat, fish, butter, eggs...) will not reduce your total cholesterol. In fact, it has been shown that reducing the carbohydrate content in a diet to 40-45% instead of 55% can significantly reduce the total cholesterol content in just a few weeks.

Increased uric acid

The uric acid level in the blood can cause deficient excretion, increased intake through food or increased self-production due to cell decay or cell conversion –. and in high concentrations, uric acid forms crystals, which can result in kidney stones or gout.

The assumption that the increased uric acid level is caused by excessive meat consumption leads to an ovo-lacto-vegetarian dietary recommendation. Low-fat milk, milk products and eggs are recommended as protein sources. Meat and meat products, fish and sausages should only be eaten in small quantities. Animal intestines, herring, mackerel and smoked sprats should generally be avoided – and poultry should not be eaten at all.

In the case of this recommendation, the external uric acid supply is generally taken into account. The individual metabolism and the body’s own production of uric acid is completely ignored. Most of the uric acid is produced by the breakdown of protein from muscle cells into glucose. This process is called gluconeogenesis, which to some extent is a normal metabolic process.

If the blood sugar level in the body cells drops, e.g. as a result of lack of food, energy can be made available to the body again by converting protein into glucose or fats into ketones. In the case of those who eat too many carbohydrates, the metabolism loses its ability to provide energy from fats. It then uses the quick and easy way to convert protein into glucose with the consequence that much more uric acid is produced.

Gluconeogenesis is the actual cause of increased uric acid levels. From this point of view, a low-carbohydrate and metabolism-optimised diet is the right way to break down uric acid.

Other causes of elevated uric acid levels:

  • medicines, such as antihypertensive drugs, acetylsalicylic acid (inhibits uric acid excretion)
  • kidney problems – addressing other kidney parameters in the blood count